Article ID Journal Published Year Pages File Type
10088747 International Journal of Pediatric Otorhinolaryngology 2005 5 Pages PDF
Abstract
The results of the present study supports that l-arginine:NO pathway may be key the participant in the pathogenesis of chronic adenotonsillar disease; arginase and iNOS activities are altered in children with adenotonsillar hypertrophy and this alteration improves after tonsillectomy.
Keywords
Related Topics
Health Sciences Medicine and Dentistry Otorhinolaryngology and Facial Plastic Surgery
Preview
The potential inflammatory role of arginase and iNOS in children with chronic adenotonsillar hypertrophy
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