| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10299102 | European Neuropsychopharmacology | 2015 | 10 Pages |
Abstract
Similar changes in Bcl-2 and MARCKS but not in PRDX2 and NPY following both Li-treatment and IMPA1 KO suggest a mechanism different than inositol-monophosphatase1 inhibition for Li׳s effect on the latter genes. The cytokine levels results suggest that the mechanism mediating Li׳s effect on the inflammatory system differs among brain regions. Only in the hippocampus the results favor the involvement of the phosphatidylinositol (PI) cycle.
Keywords
Related Topics
Life Sciences
Neuroscience
Biological Psychiatry
Authors
O. Damri, Y. Sade, L. Toker, Y. Bersudsky, R.H. Belmaker, G. Agam, A.N. Azab,