| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10306200 | Psychoneuroendocrinology | 2012 | 19 Pages |
Abstract
In the present review I summarize the available human and rodent data in order to provide a comprehensive understanding of the biphasic role of the HPA axis and/or the GC signaling during stressor exposure in terms of spontaneous colitis development.
Keywords
T helperIBDIBSICVintracerebroventricularADXTLRTGFSNSAVPTNBSDNBSn.a.LPMCChronic subordinate colony housingACTHTNFnot assessedSDRPRSCRHCSCmRNADSSPNDMPOmessenger RNAadrenalectomywater avoidance stressChronic psychosocial stressAcetic acidTrinitrobenzene sulphonic acidColitisimmunoglobulin interferonIFNinterleukinWASovercrowdingCrohn's diseaseInflammatory bowel diseaseToll-like receptormaternal separationMincluster of differentiationVersusvs.Autonomic nervous systemdextran sulphate sodiumpostnatal dayANSColorectal cancerLamina Propria Mononuclear Cellsirritable bowel syndromesympathetic nervous systemElectric shockSocial defeattumor growth factortumor necrosis factorPVNhypothalamo–pituitary–adrenal axisHPA axismyeloperoxidaseHPAnorepinephrineparaventricular nucleuscorticotrophin releasing hormonearginine vasopressinvehVehicleCRCCorticosteroneUlcerative colitisGlucocorticoidsMineralocorticoid receptorglucocorticoid receptor
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Authors
S.O. Reber,