| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10907891 | Experimental Hematology | 2005 | 10 Pages |
Abstract
These results indicate that EPOR signaling cannot substitute for G-CSFR signaling to stimulate secondary granule protein gene expression in 32D cells. In addition, STAT3 is a critical mediator of 24p3 lipocalin gene expression in these cells.
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Authors
Lei Wang, Murat O. Arcasoy, Stephanie S. Watowich, Bernard G. Forget,