| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10907906 | Experimental Hematology | 2005 | 8 Pages |
Abstract
As opposed to recent reports for c-ABL, we do not see evidence for a functional interaction between BCR-ABL and hTERT in this model system arguing against imatinib-mediated upregulation of hTERT as a crucial factor for clonal selection and disease progression of CML.
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Authors
Ulrike Hartmann, Stefan Balabanov, Patrick Ziegler, Jörg Fellenberg, Heiko van der Kuip, Justus Duyster, Hans-Peter Lipp, Carsten Bokemeyer, Lothar Kanz, Tim H. Brümmendorf,