| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10953704 | Journal of Molecular and Cellular Cardiology | 2015 | 8 Pages |
Abstract
CaMKII is activated by oxidation of methionine residues residing in the regulatory domain. Oxidized CaMKII (ox-CaMKII) is now thought to participate in cardiovascular and pulmonary diseases and cancer. This invited review summarizes current evidence for the role of ox-CaMKII in disease, considers critical knowledge gaps and suggests new areas for inquiry.
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Authors
Mark E. Anderson,