Article ID Journal Published Year Pages File Type
10953707 Journal of Molecular and Cellular Cardiology 2015 8 Pages PDF
Abstract
Increased ROS production in the hearts of mdx mice drives the progression of cardiac dysfunction. Inhibition of RyR2 phosphorylation can suppress SR Ca2 + leak in mdx mouse hearts in part by reducing RyR2 oxidation.
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Life Sciences Biochemistry, Genetics and Molecular Biology Cell Biology
Preview
Crosstalk between RyR2 oxidation and phosphorylation contributes to cardiac dysfunction in mice with Duchenne muscular dystrophy
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