Article ID Journal Published Year Pages File Type
10953927 Journal of Molecular and Cellular Cardiology 2012 9 Pages PDF
Abstract
► Previous work has shown high dietary fat (SAT) is cardioprotective post-infarction. ► Energy metabolism changes were evaluated as the potential cardioprotective mechanism. ► SAT increases fatty acid oxidation and reduces glucose oxidation post-MI. ► Pyruvate dehydrogenase regulation is also affected, distinct from SAT or MI alone. ► We conclude SAT after MI improves lipid metabolism to confer cardioprotection.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Cell Biology
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