| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 11018571 | Pancreatology | 2018 | 11 Pages |
Abstract
Cantharidin can sensitize pancreatic cancer cells to radiotherapy. Multiple mechanisms, including cell cycle regulation, enhanced DNA damage, and inhibited DNA damage repair, may be involved.
Keywords
ATRMMRPARPBERXRCC1PP2ANHEJHRRFANCD2DSBFANCIataxia telangiectasia mutatedDMSONERMTTDNA damage repairnucleotide excision repairHomologous recombination repairmismatch repairbase excision repairATMDimethyl sulfoxideRadiotherapyPancreatic cancerdouble-strand breakmethyl thiazolyl tetrazoliumHomologous recombinationprotein phosphatase 2Apoly-ADP-ribose polymeraseNonhomologous end-joiningCell cycleCantharidinFanconi anemia
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Authors
Meng-Dan Xu, Shu-Ling Liu, Bei-Bei Zheng, Jing Wu, Meng-Yao Wu, Yan Zhang, Fei-Ran Gong, Min Tao, Junning Zhang, Wei Li,