| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 11028751 | Life Sciences | 2018 | 14 Pages |
Abstract
In conclusion, AR-42 may increase the sensitivity of CML cells to IM and reverse IM resistance by regulating HDAC1 expression. This study provides new insights into the effects of combined therapy using IM and pan-HDAC inhibitor AR-42, paving the way for overcoming IM resistance in clinical practice.
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Authors
Danna Wei, Tingting Lu, Dan Ma, Kunlin Yu, Tianzhuo Zhang, Jie Xiong, Weili Wang, Zhaoyuan Zhang, Qin Fang, Jishi Wang,