| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 11033796 | Metabolism | 2018 | 10 Pages |
Abstract
7,8-DHF treatment increases muscular mitochondrial respiration and systemic energy expenditure, which alleviates the body weight gain and partially reverse the metabolic abnormalities induced by obesity.
Keywords
VMHADIPOQTrkBPKCAMPK7,8-DHFcyto cPLCγTFAMNRF1VDACangiopoietin-like 3PGC-1αHGFM-CSFsdhARERPDHIGFBPTIMP1IWATTropomyosin-related kinase receptor Bsuccinate dehydrogenase complex subunit AFFACREBACCBATDMHHFDPPARγ7,8-DihydroxyflavoneAMP-activated protein kinaseBDNFIgf1Ucp1adiponectinFree fatty acidFatty acid oxidationinguinal white adipose tissuebrown adipose tissuetriglycerideHigh fat dietcytochrome cNuclear respiratory factor 1Hepatocyte growth factormitochondrial transcription factor Abrain derived neurotrophic factorSkeletal muscleFAOVascular endothelial growth factorVascular Endothelial Growth Factor (VEGF)insulin-like growth factor 1macrophage colony-stimulating factorObesityMitochondrialRespiratory exchange ratiodorsomedial hypothalamusinsulin-like growth factor binding proteincAMP responsive element binding proteinuncoupling protein 1C-reactive proteinCRPProtein kinase Cpyruvate dehydrogenaseinsulin receptorperoxisome proliferator-activated receptor γ
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Authors
John Wood, Margaret Chui Ling Tse, Xiuying Yang, Daniel Brobst, Zhixue Liu, Brian Pak Shing Pang, Wing Suen Chan, Aung Moe Zaw, Billy K.C. Chow, Keqiang Ye, Chi Wai Lee, Chi Bun Chan,