| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 11033827 | Cytokine | 2018 | 7 Pages |
Abstract
Our data suggest that local or systemic immunomodulatory mediators are unsuitable for predicting the disease course of BA. Thus, no deduction for optimal treatment strategy can be drawn. Collectively, we conclude that in BA, the degree of inflammation and protein microenvironment in the liver at the time-point of KPE are dismissible factors for the future course of disease.
Keywords
Vascular endothelial growth factor alphaVEGF-APIGFCCLLTXSCFCXCLHGFGM-CSFTGFKPEIL-1RARLCSNLFGFSDFEGFLIFNCBITNFPDGF-BBMCPCC-chemokine ligandMIPBDNFPCAinterleukin-1 receptor antagonistenzyme linked immunosorbent assaystandard deviationBiliary atresiainterferonIFNinterleukinBARDPrincipal component analysisELISAliver cirrhosisFigfigureStromal cell-derived factorepidermal growth factortumor growth factorplacenta growth factorHepatocyte growth factorleukemia inhibitory factorTumor necrosis factorsfibroblast growth factorStem Cell FactorBrain-derived neurotrophic factorgranulocyte macrophage colony-stimulating factorMedical productsNational Center for Biotechnology InformationRANTESLong term outcomemacrophage inflammatory proteinsmonocyte chemoattractant proteinLiver transplantationplatelet-derived growth factor receptor
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Authors
Omid Madadi-Sanjani, Joachim F. Kuebler, Stephanie Dippel, Anna Gigina, Christine S. Falk, Gertrud Vieten, Claus Petersen, Christian Klemann,