| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 1904914 | Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease | 2012 | 14 Pages |
Abstract
⺠ACOTs regulate acyl-CoA metabolism in peroxisomes. ⺠Peroxisomes produce glycine and taurine-conjugated fatty acids. ⺠BAAT is involved in the inheritance of familial hypercholanemia. ⺠New insights into acyl-CoA thioesterase structure and function. ⺠ACOTs structurally belong to the α/β hydrolase and HotDog fold protein superfamilies.
Keywords
4-HNE4-hydroxynonenalNATPPARBAATBile acid-CoA:amino acid N-acyltransferaseFAAHPTS1CDCAAcotFXRHNF-4αacyl-CoA thioesterasefarnesoid X receptorChenodeoxycholic acidFatty acid amide hydrolaseCholic acidProtein structurehepatocyte nuclear factor 4 alphaperoxisome coenzyme ACoASHPeroxisome proliferator activated receptor
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Authors
Mary C. Hunt, Marina I. Siponen, Stefan E.H. Alexson,