Deletion of GSTA4-4 results in increased mitochondrial post-translational modification of proteins by reactive aldehydes following chronic ethanol consumption in mice

Article ID	Journal	Published Year	Pages	File Type
1922837	Redox Biology	2016	10 Pages	PDF

Keywords

4-HNE 4-oxononenal 4-ONE gsta4 MDA 4-HHE CYP2E1 ADPh ALT CID ETD EtOH 4-hydroxy-2-nonenal 4-Hydroxy-2-hexenal ALD Adipophilin Alanine aminotransferase Ethanol Collision-induced dissociation Electron transfer dissociation alcoholic liver disease Oxidative stress pair-fed cytochrome P4502E1 malondialdehyde Mitochondria Lipid peroxidation Liver Protein carbonylation

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Ageing

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Deletion of GSTA4-4 results in increased mitochondrial post-translational modification of proteins by reactive aldehydes following chronic ethanol consumption in mice

Authors

Colin T. Shearn, Kristofer S. Fritz, Alisabeth H. Shearn, Laura M. Saba, Kelly E. Mercer, Bridgette Engi, James J. Galligan, Piotr Zimniak, David J. Orlicky, Martin J. Ronis, Dennis R. Petersen,