| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2172743 | Cytotherapy | 2010 | 7 Pages |
Abstract
We found that adoptive transfer of PR1-CTL generated in vitro is associated with reduced AML cells in NOD/SCID mice. PR1-CTL can migrate to the sites of disease and maintain their capacity to kill the AML cells. The surface phenotype of PR1-CTL was consistent with their trafficking pattern in both vascular and end-organ tissues.
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Authors
Qing Ma, Changqing Wang, Dan Jones, Kathryn E. Quintanilla, Dan Li, Yang Wang, Eric D. Wieder, Karen Clise-Dwyer, Gheath Alatrash, You Mj, Mark F. Munsell, Sijie Lu, Muzaffar H. Qazilbash, Jeffrey J. Molldrem,