| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2182600 | Genes & Diseases | 2016 | 4 Pages |
Abstract
Two recent studies provide intriguing evidence that challenges the role of the epithelial–mesenchymal transition (EMT) as a critical mediator of cancer metastasis, while revealing an unexpected role in cancer drug resistance.1,2 While these findings may not settle the EMT's role in metastasis, these studies suggest that targeting the EMT may inhibit both cancer metastasis and chemoresistance.
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Authors
Jing Wang, Qiang Wei, Xin Wang, Shengli Tang, Hao Liu, Fugui Zhang, Maryam K. Mohammed, Jiayi Huang, Dan Guo, Minpeng Lu, Feng Liu, Jianxiang Liu, Chao Ma, Xue Hu, Rex C. Haydon, Tong-Chuan He, Hue H. Luu,