Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
2790274 | Placenta | 2006 | 6 Pages |
Abstract
Increased PAF-AH activity in trophoblasts may be a compensatory effect to control PAF level in the preeclamptic placenta. The co-existence of PAF-AH and PAF receptor in trophoblasts suggests an autocrine regulation of PAF in these cells to limit PAF and its metabolites within the placenta.
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Authors
Y. Gu, S.A. Burlison, Y. Wang,