Article ID Journal Published Year Pages File Type
2790274 Placenta 2006 6 Pages PDF
Abstract
Increased PAF-AH activity in trophoblasts may be a compensatory effect to control PAF level in the preeclamptic placenta. The co-existence of PAF-AH and PAF receptor in trophoblasts suggests an autocrine regulation of PAF in these cells to limit PAF and its metabolites within the placenta.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Developmental Biology
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