Article ID Journal Published Year Pages File Type
2864053 The American Journal of the Medical Sciences 2011 6 Pages PDF
Abstract
Increased ER stress might be responsible for enhanced cardiomyocyte apoptosis after aortic coarctation. Telmisartan may reduce ER stress and thereby attenuate both apoptosis and cardiac hypertrophy.
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Related Topics
Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine
Preview
Endoplasmic Reticulum Stress Caused by Left Ventricular Hypertrophy in Rats: Effects of Telmisartan
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