| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2864954 | The American Journal of the Medical Sciences | 2008 | 8 Pages |
Abstract
The current study suggests that iNOS-induced nitric oxide production may not mediate cardiac fibrosis after MI. Thus, other mechanisms are involved in nitrosative stress-induced cardiac dysfunction after MI.
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Authors
Li MD, Sue Si BA, Aviv PhD, Yao MD, PhD,