| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2872681 | The Annals of Thoracic Surgery | 2015 | 6 Pages |
Abstract
Ischemia-reperfusion injury induces EPO receptor βcR subunit expression and early downstream anti-apoptotic signaling through STAT3 activation. Further investigation into the role of the βcR subunit is warranted to determine tissue protective functions of EPO. Elucidation of mechanisms involved in spinal cord protection is essential for reducing delayed paraplegia.
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Authors
Lisa S. MD, David A. MD, Daine T. MD, Kirsten A. MD, Joshua BA, Marshall T. MD, Joseph C. MD, Michael J. MD, Xianzhong PhD, MD, Ferenc MD, PhD, T. Brett MD,