| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2879289 | The Annals of Thoracic Surgery | 2008 | 6 Pages |
Abstract
The alveolar macrophage drives the development of lung reperfusion injury in part through amplification of the inflammatory response of type 2 pneumocytes subjected to hypoxia and reoxygenation.
Keywords
MIP-2PBST2PMIP-1αJnkFBSIL-1βPAECphosphate buffered saline with TweenPBSTMCP-1RPMIc-Jun N-terminal kinaseERK1/2MAPKNFκBLIRIElectrophoretic mobility shift assayenzyme linked immunosorbent assayInterleukin-1 betaELISAtumor necrosis factor alphafetal bovine serumpulmonary artery endothelial cellEMSA یا electrophoretic mobility shift assay TNF-αnuclear factor kappa BPhosphate buffered salineAlveolar macrophagemacrophage inflammatory protein-2macrophage inflammatory protein-1 alphamajor histocompatibility complexMHChypoxia/reoxygenationMonocyte chemotactic protein-1mitogen-activated protein kinaseCINCextracellular signal-regulated kinase
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Authors
Anton S. MRCS, Alexander S. MD, Steven M. MRCS, Heather E. MD, Patrick S. MD, Brendan BS, John C. MD, Elizabeth MD, Michael S. MD,