| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2893659 | Atherosclerosis | 2010 | 7 Pages |
Abstract
In hyperhomocysteinemia increases in flow/shear stress increases the production of COX-2-derived TxA2, and reactive oxygen species - that overcome the dilator effects of NO and prostaglandins - eliciting constrictions in skeletal muscle venules; changes which can increase vascular resistance and favor thrombus formation in the venular circulation.
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Authors
Anita Racz, Zoltan Veresh, Gabor Lotz, Zsolt Bagi, Akos Koller,