| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2894737 | Atherosclerosis | 2008 | 7 Pages |
Abstract
In this group of patients with FCHL, the major defect appeared to be overproduction of TAG by the liver due to decreased fatty acid oxidation, with fatty acids directed to TAG synthesis. We found no evidence of decreased lipoprotein lipase action or impaired fatty acid re-esterification in adipose tissue.
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Authors
Kevin Evans, Graham C. Burdge, Stephen A. Wootton, Jenny M. Collins, Mo L. Clark, Garry D. Tan, Fredrik Karpe, Keith N. Frayn,