Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
2936101 | International Journal of Cardiology | 2006 | 4 Pages |
Abstract
A 57-year-old man came to the emergency room because of abrupt chest pain for 30Â min. ST elevations in inferior lead were normalized following sublingual nitroglycerin. He performed an exercise test on Bruce protocol and exercised up to 12Â min (13 METs) without chest pain and ST-T changes on the next day. We performed coronary angiography to rule out coronary vasospasm; however, we did not perform provocation test due to thombus in the culprit lesion. After medical treatment for 6Â days, follow-up coronary angiogram revealed no thrombus in the previous site and intravascular ultrasound did not demonstrate plaque rupture. We concluded that prolonged spasm might cause prolonged coronary flow limitation and induce acute thrombus formation without plaque rupture. This finding may be an important pathogenesis of acute coronary syndrome in patients with variant angina.
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Authors
Seung Hwan Han, Kwang Kon Koh, Kyu Jin Oh, Kyu Hyun Yoon,