| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2936243 | International Journal of Cardiology | 2007 | 7 Pages |
Abstract
Ang II and ET-1 elicited PIE in atrial myocytes, whereas in ventricular myocytes Ang II did not induce PIE in contrast to ET-1. The PIE of ET-1 was markedly attenuated in failing myocytes. Under acidotic conditions, the PIE of ET-1 was more pronounced in failing myocytes, indicating that ET-1 may activate signaling processes in failing myocytes, which are not activated in normal myocytes.
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Authors
Helge Möllmann, Stephan Schmidt-Schweda, Holger Nef, Susanne Möllmann, Johannes V. Burstin, Stefan Klose, Albrecht Elsässer, Christian J.F. Holubarsch,