| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3042816 | Clinical Neurophysiology | 2015 | 8 Pages |
Abstract
The mechanisms underlying the generation of slow EEG components may differ between ES and IIED. The slow EEG components of ES might indicate an extensive epileptic network involving remote symptomatic zones for ES in either the cortical or subcortical areas. The high spatial concordance between the fast and slow components of IIED suggests the involvement of a local inhibitory process within the epileptic cortex.
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Authors
Tomoyuki Akiyama, Mari Akiyama, Katsuhiro Kobayashi, Tohru Okanishi, Cyrus G. Boelman, Dragos A. Nita, Ayako Ochi, Cristina Y. Go, O Carter III, James T. Rutka, James M. Drake, Sylvester Chuang, Hiroshi Otsubo,