| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3152474 | Journal of Oral and Maxillofacial Surgery | 2015 | 5 Pages |
Abstract
Non-medication–related acquired deficiencies of C1 esterase inhibitor (C1-INH) can cause the facial acquired angioedema (AAE) seen in systemic lupus erythematosus (SLE). The defect can originate from a lymphoproliferative disease (LPD) that catabolizes C1-INH or from circulating antibodies that inactivate C1-INH. This report describes a third and rare variety of facial AAE originating in SLE in which there was no LPD or circulating antibodies to impede C1-INH activity.
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Authors
Daniel Bienstock, Louis Mandel,