| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3198187 | Journal of Allergy and Clinical Immunology | 2013 | 28 Pages |
Abstract
Gain-of-function mutations in STAT1 predispose to invasive, severe, disseminated dimorphic yeast infections, likely through aberrant regulation of IFN-γ-mediated inflammation.
Keywords
PMLISREL-AMBNIHPIASIFN-γpStat1ThrushGFPgamma-activated sequencePhosphorylated STAT1DMACMCCoccidioides immitisSmall interfering RNAsiRNAStat1Liposomal amphotericin BS-adenosylmethionineMRIMagnetic resonance imagingcomputed tomographyProgressive multifocal leukoencephalopathyNIH, National Institutes of HealthCSFCerebrospinal fluidsignal transducer and activator of transcription 1protein inhibitor of activated Statwild-typeHistoplasma capsulatumWestern blottinggreen fluorescent proteinchronic mucocutaneous candidiasisGasSAMe
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Authors
Elizabeth P. MD, PhD, Amy P. BA, Joseph BA, Hannelore I. MD, Dalton L. BA, Michelle L. MD, Prabha PhD, Lindsey B. BS, Daniel S. PhD, Li MD, Donald C. MD, Sarah K. MD, Shrimati PhD, Joshua D. MD, Douglas B. PhD, Debra A. BS, Mohammed A. MD, Michael MD,