Article ID Journal Published Year Pages File Type
3198501 Journal of Allergy and Clinical Immunology 2012 11 Pages PDF
Abstract
Our data indicate that epithelial IRAK-M overexpression in TH2 cytokine-exposed airways inhibits TLR2 signaling, providing a novel mechanism for the increased susceptibility of infections in asthmatic patients.
Related Topics
Life Sciences Immunology and Microbiology Immunology
Authors
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