| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3201471 | Journal of Allergy and Clinical Immunology | 2008 | 9 Pages |
Abstract
The increase of plasma fibrinogen levels, its vasodilator activity in human ITAs, and the potentiation of bradykinin-induced vasodilation suggest that fibrinogen might contribute to the pathophysiology of ACEi-induced angioedema. Thus acute-phase proteins, such as fibrinogen, might be viewed as risk factors for bradykinin-induced angioedema.
Keywords
SPCAITAVASPATP-sensitive potassiumVSMKATPPGF2αeNOSANCOVAcGMPl-NAMEAngioedemaAngiotensin converting enzyme inhibitorbradykininanalysis of covarianceendothelial nitric oxide synthaseVasodilator-stimulated phosphoproteinphenylephrineFibrinogenangiotensin-converting enzyme inhibitorACEI یا angiotensin convert enzyme inhibitorcyclic guanosine monophosphateProstaglandin F2α
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Authors
Murat MD, Nadine Kirchhartz, Jessica Hochfeld, Cornelia Tüllmann, Stephanie Kumpf, Tatsiana PhD, Marc PharmD, Dieter PhD, Henning MD, Thomas K. MD, Vera PhD, Georg PharmD, PhD,