| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3293555 | Gastroenterology | 2012 | 33 Pages |
Abstract
In mice, reversal of HSC activation contributes to termination of fibrogenesis during fibrosis resolution, but results in higher responsiveness of reverted HSCs to recurring fibrogenic stimulation.
Keywords
ECMHSCPDGFFACSCCl4TAATGFβFBSqPCRα smooth muscle actinThioacetamidefluorescence-activated cell sortingfetal bovine serumHepatic stellate cellCirrhosisplatelet-derived growth factorExtracellular matrixMouse modelMyofibroblasthepatitisquantitative real-time polymerase chain reactionCarbon tetrachloride
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Authors
Juliane S. Troeger, Ingmar Mederacke, Geum-Youn Gwak, Dianne H. Dapito, Xueru Mu, Christine C. Hsu, Jean-Philippe Pradere, Richard A. Friedman, Robert F. Schwabe,