| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3293930 | Gastroenterology | 2010 | 34 Pages |
Abstract
Polymorphisms in genes involved in the regulation of angiogenesis are associated with the risk of HPS. Further investigation of these biologic pathways might elucidate the mechanisms that mediate the development of HPS in certain patients with severe liver disease.
Keywords
Runx1NOX4PaO2HPSEndoglinCAV3ENGCARTSHBGHWEESR2vWFepidermal growthSat2Estrogen receptor 2Tie1EGF95% CIHIF1aNADPH oxidase 4Hardy–Weinberg equilibriumHepatopulmonary syndromeClassification and regression treesrunt-related transcription factor 1Von Willebrand factor95% confidence intervalpartial pressure of oxygen in arterial bloodModel for End-Stage Liver Diseaseodds ratioAncestry informative markerAIMPortal hypertensionGenetic polymorphismSingle nucleotide polymorphismSNPcaveolin 3MELD
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Authors
Kari E. Roberts, Steven M. Kawut, Michael J. Krowka, Robert S. Jr, James F. Trotter, Vijay Shah, Inga Peter, Hocine Tighiouart, Nandita Mitra, Elizabeth Handorf, James A. Knowles, Steven Zacks, Michael B. Fallon,