Article ID Journal Published Year Pages File Type
3295297 Gastroenterology 2009 19 Pages PDF
Abstract
CcnE2 has an unexpected role in repressing CcnE1; the phenotype of E2−/− mice appears to result from CcnE1 overexpression and cdk2 hyperactivation. CcnE1 and CcnE2 therefore have nonredundant functions for S-phase entry and endoreplication during liver regeneration.
Related Topics
Health Sciences Medicine and Dentistry Gastroenterology
Authors
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