| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3296166 | Gastroenterology | 2011 | 12 Pages |
Abstract
Epistatic and biological interactions between TLE1 and NOD2 are involved in IBD pathogenesis. NOD2 might be involved in a series of pathways such as epigenetic regulation of expression (via TLE1 and HTATIP), biosynthesis of mucin (via GALNT2), apoptosis (via PPP2R5E and FIS1), and integrity of the intracellular cytoskeleton (vimentin).
Keywords
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Authors
Elaine R. Nimmo, Craig Stevens, Anne M. Phillips, Amanda Smith, Hazel E. Drummond, Colin L. Noble, Michael Quail, Gail Davies, Marian C. Aldhous, David C. Wilson, Jack Satsangi,