| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3296532 | Gastroenterology | 2010 | 13 Pages |
Abstract
The down-regulation of KL that occurs during inflammation might account for the extraintestinal complications such as abnormalities in bone homeostasis that occur in patients with IBD.
Keywords
S-nitroso-N-acetyl-dl-penicillamineiNOSIBDRT-PCRFGF23mRNATBPTNBSSPFTRPV5JnkTNFPBSc-Jun N-terminal kinasereverse-transcription PCRmessenger RNATrinitrobenzene sulfonic acidinterferonIFNinterleukinInflammatory bowel diseaseELISAEnzyme-linked immunosorbent assayforkhead Box Oinducible nitric oxide synthaseSNAPnuclear factorfibroblast growth factor 23tumor necrosis factorFoxOdistal convoluted tubulesBone metabolismPhosphate-buffered salinewild typeNitric oxideMineral homeostasispolymerase chain reactionPCRspecific pathogen freeTATA-box binding proteinKlothoKidney
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Authors
Robert D. Thurston, Claire B. Larmonier, Pawel M. Majewski, Rajalakshmy Ramalingam, Monica Midura-Kiela, Daniel Laubitz, Alain Vandewalle, David G. Besselsen, Marcus Mühlbauer, Christian Jobin, Pawel R. Kiela, Fayez K. Ghishan,