| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3316776 | Pancreatology | 2014 | 7 Pages |
Abstract
FliC from E. coli may pathogenetically generate an AIP-like inflammation status. A reconsideration of the importance of commensal bacteria as an environmental factor(s) capable of inducing autoimmunity could provide insight to overcoming AIP.
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Authors
Naoko Yanagisawa, Ikuko Haruta, Kyoko Shimizu, Toru Furukawa, Tomoaki Higuchi, Noriyuki Shibata, Keiko Shiratori, Junji Yagi,