| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3317176 | Pancreatology | 2013 | 7 Pages |
Abstract
Our analyses clearly demonstrate that HBPs form disease-specific and highly connected networks that can be explored for potential biomarkers and as collective drug targets via the modification of heparin binding properties.
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Authors
Q.M. Nunes, V. Mournetas, B. Lane, R. Sutton, D.G. Fernig, O. Vasieva,