Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
342204 | Seizure | 2010 | 4 Pages |
Abstract
We describe the ictal [18F]FDG-PET study of a case of absence status showing bilateral thalamic hypermetabolism and frontal cortex hypometabolism. This is the first ictal assessment of absence status by [18F]FDG-PET reporting this particular cortical and subcortical involvement. Our findings support the theory of corticothalamic circuitry involvement in the pathophysiology of absence seizures and stress the similarities of the clinical and metabolic pattern observed during absences with the pattern of task-induced interruption of the default state of brain function.
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Authors
Leonilda Bilo, Roberta Meo, Maria Fulvia de Leva, Caterina Vicidomini, Marco Salvatore, Sabina Pappatà ,