Article ID Journal Published Year Pages File Type
3433730 American Journal of Obstetrics and Gynecology 2012 5 Pages PDF
Abstract
Prenatal alcohol exposure acutely results in a significant elevation of IL-6, G-CSF and the KC, which are known to affect N-methyl-D-aspartate receptors. NAP plus SAL treatment prevented alcohol-induced increases. This provides additional insight into the mechanism of alcohol damage in FAS and NAP plus SAL prevention of neurodevelopmental anomalies.
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Neuroprotective peptides influence cytokine and chemokine alterations in a model of fetal alcohol syndrome
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