Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
3436775 | American Journal of Obstetrics and Gynecology | 2011 | 7 Pages |
Abstract
The reduced endothelial nitric oxide and inducible nitric oxide synthases gene expression in women with hemolysis, elevated liver enzyme levels, and low platelet count syndrome may indicate extreme placental dysfunction that is unable to compensate the endothelial derangement and the related hypertension. The higher nitric oxide formation found in hemolysis, elevated liver enzyme levels, and low platelet count syndrome placentas could be explained as a counteraction to the impaired fetoplacental perfusion, typical of the syndrome.
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Authors
Laura Mazzanti, Monia PhD, Arianna PhD, Sara MD, Monica Emanuelli, Stefano R. MD, PhD, Franca Saccucci, Andrea L. MD,