Article ID Journal Published Year Pages File Type
3437975 American Journal of Obstetrics and Gynecology 2008 4 Pages PDF
Abstract
Our data do not support suppression of TLR pathways as a mechanism for the benefit of 17-α hydroxyprogesterone caproate. Increased CASP8 gene expression raises the possibility that progestins “prime” the decidual cell to respond with a NFκB-mediated inflammatory response.
Keywords
Related Topics
Health Sciences Medicine and Dentistry Medicine and Dentistry (General)
Preview
Human decidual cell Toll-like receptor signaling in response to endotoxin: The effect of progestins
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