| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3850786 | American Journal of Kidney Diseases | 2009 | 12 Pages |
Abstract
Evaluation of human AKI revealed early intraoperative tubular stress in all patients. A second phase of injury observed in patients with AKI may involve IP-10 recruitment of inflammatory cells. The enhancement of hepcidin-25 in patients without AKI may suggest a novel role for iron sequestration in modulating AKI.
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Authors
Julie MD, Malcolm BSc, Oleg PhD, Kent PhD, Edward MD, Gayle RN, David MD, Peter MD, Claudio MD, Martina MD, PhD,