| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3869534 | The Journal of Urology | 2010 | 8 Pages |
Abstract
Olfactory mucosal transplantation after spinal cord injury weakened external urethral sphincter excessive bursting and increased the urethral opening to improve voiding efficiency. Olfactory mucosal transplantation may modify emergence of the spinal micturition reflex after spinal cord injury. Transplantation resulted in new axons growing at the transplant site, implying the possible existence of interneuron bridging across the injured spinal cord.
Keywords
neurofilament MEUSPBSGap43OMTCGRPTUJ-1HFOOECCMGVCDNFMGFAPDSDvoiding efficiencySpinal cord injuriesSpinal cord injuryUrination disordersexternal urethral sphincterEMGelectromyogramICIpost-void residual urine volumegrowth associated protein 43olfactory ensheathing cellCystometrogramPVRsciPhosphate buffered salineIntravesical pressureUrethraOlfactory mucosaHigh frequency oscillationGlial fibrillary acidic proteincalcitonin gene-related peptideTransplantation
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Authors
Jiro Nakayama, Tetsuya Takao, Hiroshi Kiuchi, Keisuke Yamamoto, Shinichiro Fukuhara, Yasushi Miyagawa, Masanori Aoki, Koichi Iwatsuki, Toshiki Yoshimine, Masaki Ueno, Toshihide Yamashita, Norio Nonomura, Akira Tsujimura, Akihiko Okuyama,