Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
3878130 | The Journal of Urology | 2006 | 7 Pages |
Abstract
NHBD renal transplants were accompanied by a greater release of free radicals at reperfusion (NHBD > HBD > LD), which was associated with an increase in tissue injury markers at reperfusion. This was reflected in a slower return to stable renal function in NHBD compared to HBD and LD renal transplants.
Keywords
H-FABPDGFPBNHbdNHBDα-phenyl-N-tert-butyl nitroneFABPESRASTAspartate aminotransferaseReperfusion injuryAlanine aminopeptidaseLiving donortissue donorsIschemiaischemia-reperfusiondelayed graft functionELISAEnzyme-linked immunosorbent assayElectron spin resonancelactate dehydrogenaseLDHNADHFatty acid binding proteinKidney transplantationreduced nicotinamide adenine dinucleotideKidney
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Authors
M.A. Gok, B.K. Shenton, M. Pelsers, A. Whitwood, D. Mantle, C. Cornell, R. Peaston, D. Rix, B.C. Jaques, N.A. Soomro, D.M. Manas, D. Talbot,