| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3887209 | Kidney International | 2007 | 4 Pages |
Abstract
Diabetic nephropathy is currently viewed as a predominantly glomerular process with glomerular injury driving secondary tubular loss. Brezniceanu and colleagues apply transgenic methods to support a prominent role for reactive oxygen species as mediators and for the proximal tubule as a major site of early disease activity in diabetes. Results support evidence for early tubular apoptosis and atrophy in human diabetic nephropathy.
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Authors
S.-P. Bagby,