| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3902668 | Urology | 2011 | 7 Pages |
Abstract
Our findings suggest that VDR knockdown in the kidney can upregulate the expression of transient receptor potential vanilloid receptor subtype 5 in GHS rats. However, VDR depletion results in an increase in urine calcium excretion. The role of VDR in the hypercalciuric formation needs to be elucidated further.
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Authors
Qi-Lin Xi, Shao-Gang Wang, Zhang-Qun Ye, Zhao-Wei Zhu, Cong Li, Jian Bai, Xiao Yu, Ji-Hong Liu,