| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 3940373 | Fertility and Sterility | 2006 | 5 Pages |
Abstract
Isolated hypogonadotropic hypogonadism may result from an acquired defect of enhanced hypothalamic sensitivity to E-mediated negative feedback. Whereas direct T replacement therapy can further suppress endogenous gonadotropin secretion, treating IHH men with gonadotropins can stimulate endogenous T secretion and enhance fertility potential. On theoretical grounds, reversal of gonadotropin deficiency with CC might be expected to have a similar biological effect.
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Authors
Stella M.D., Pat J. M.B., Ch.B., R. Dermot M.D., Richard M.D.,