Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
3990906 | Journal of Thoracic Oncology | 2010 | 6 Pages |
Abstract
The occurrence of MET amplification and EGFR/ K-ras mutations might be mutually exclusive suggesting several distinct mechanisms in the development of lung adenocarcinoma. The wild type of K-ras, negative p-MET 1234/1235, and positive expression of HGF may be a useful marker for predicting poor prognosis of patients who underwent surgical resection of lung adenocarcinoma.
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Authors
Takamitsu MD, Hidetaka PhD, MD, Kenji PhD, MD, Mitsuhiro PhD, MD, Takeshi PhD, MD, Tsunehiro PhD, MD, Hiroto PhD, MD, Kimitoshi PhD, MD, Kosei PhD, MD,