| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4104495 | American Journal of Otolaryngology | 2007 | 7 Pages |
Abstract
BMS-275183 induced cellular apoptosis, cell-cycle arrest, and altered gene expression in HNSCC via molecular pathways similar to other taxanes. These preclinical experiments suggest that BMS-275183 may be useful in treating HNSCC and that the aforementioned genes can potentially be used as surrogate end-point biomarkers.
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Authors
George H. MD, Vivian R. MD, Lori A. MD, Waleed H. MD, Geetha MD, Marie P. PhD, John F. MD, Fulvio MD, Harold MD, Ho-Sheng MD,