| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4177346 | Biological Psychiatry | 2014 | 8 Pages |
Abstract
Our data provide evidence that disruption of p11 homeostasis in the NAc, particularly in dopamine receptor-1 expressing medium spiny neurons, may underlie pathophysiological mechanisms of cocaine rewarding action. Treatments to counter maladaptation of p11 levels may provide novel therapeutic opportunities for cocaine addiction.
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Authors
Margarita Arango-Lievano, Justin T. Schwarz, Mary Vernov, Matthew B. Wilkinson, Kathryn Bradbury, Akira Feliz, Roberta Marongiu, Yaroslav Gelfand, Jennifer Warner-Schmidt, Eric J. Nestler, Paul Greengard, Scott J. Russo, Michael G. Kaplitt,